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Clarification of the potential role of CSF-1 in activation of macrophages.

Journal of leukocyte biology

Evans R.
PMID: 1856602
J Leukoc Biol. 1991 Sep;50(3):316. doi: 10.1002/jlb.50.3.316.

No abstract available.

Cite
Evans R. Clarification of the potential role of CSF-1 in activation of macrophages. J Leukoc Biol. 1991;50(3):316doi: 10.1002/jlb.50.3.316.
Evans, R. (1991). Clarification of the potential role of CSF-1 in activation of macrophages. Journal of leukocyte biology, 50(3), 316. https://doi.org/10.1002/jlb.50.3.316
Evans, R. "Clarification of the potential role of CSF-1 in activation of macrophages." Journal of leukocyte biology vol. 50,3 (1991): 316. doi: https://doi.org/10.1002/jlb.50.3.316
Evans R. Clarification of the potential role of CSF-1 in activation of macrophages. J Leukoc Biol. 1991 Sep;50(3):316. doi: 10.1002/jlb.50.3.316. PMID: 1856602.
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Editorial: Flexible Syk: turning on and off the inflammasome as needed.

Journal of leukocyte biology

Fitzpatrick E.
PMID: 28319938
J Leukoc Biol. 2015 May;97(5):821-824. doi: 10.1189/jlb.2CE1214-627R.

No abstract available.

Cite
Fitzpatrick E. Editorial: Flexible Syk: turning on and off the inflammasome as needed. J Leukoc Biol. 2015;97(5):821-824doi: 10.1189/jlb.2CE1214-627R.
Fitzpatrick, E. (2015). Editorial: Flexible Syk: turning on and off the inflammasome as needed. Journal of leukocyte biology, 97(5), 821-824. https://doi.org/10.1189/jlb.2CE1214-627R
Fitzpatrick, Elizabeth. "Editorial: Flexible Syk: turning on and off the inflammasome as needed." Journal of leukocyte biology vol. 97,5 (2015): 821-824. doi: https://doi.org/10.1189/jlb.2CE1214-627R
Fitzpatrick E. Editorial: Flexible Syk: turning on and off the inflammasome as needed. J Leukoc Biol. 2015 May;97(5):821-824. doi: 10.1189/jlb.2CE1214-627R. PMID: 28319938.
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Erratum.

Journal of leukocyte biology

[No authors listed]
PMID: 29485760
J Leukoc Biol. 2018 Mar;103(3):603. doi: 10.1002/jlb.10110.

No abstract available.

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Erratum. J Leukoc Biol. 2018;103(3):603doi: 10.1002/jlb.10110.
(2018). Erratum. Journal of leukocyte biology, 103(3), 603. https://doi.org/10.1002/jlb.10110
"Erratum." Journal of leukocyte biology vol. 103,3 (2018): 603. doi: https://doi.org/10.1002/jlb.10110
Erratum. J Leukoc Biol. 2018 Mar;103(3):603. doi: 10.1002/jlb.10110. PMID: 29485760; PMCID: PMC6988894.
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Prostaglandins produced during class A scavenger receptor-mediated macrophage adhesion differentially regulate cytokine production.

Journal of leukocyte biology

Nikolic DM, Vadali S, He B, Ware J, Kelly T, Post SR.
PMID: 25717147
J Leukoc Biol. 2015 May;97(5):901-908. doi: 10.1189/jlb.2A1014-471RR. Epub 2015 Feb 25.

Inflammation is associated with modification of the extracellular environment, changes in cytokine expression, and the accumulation of immune cells. Such modifications create ligands that support SR-A-mediated macrophage adhesion and retention. This may be particularly important in settings, such as...

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Nikolic DM, Vadali S, He B, et al. Prostaglandins produced during class A scavenger receptor-mediated macrophage adhesion differentially regulate cytokine production. J Leukoc Biol. 2015;97(5):901-908doi: 10.1189/jlb.2A1014-471RR.
Nikolic, D. M., Vadali, S., He, B., Ware, J., Kelly, T., & Post, S. R. (2015). Prostaglandins produced during class A scavenger receptor-mediated macrophage adhesion differentially regulate cytokine production. Journal of leukocyte biology, 97(5), 901-908. https://doi.org/10.1189/jlb.2A1014-471RR
Nikolic, Dejan M, et al. "Prostaglandins produced during class A scavenger receptor-mediated macrophage adhesion differentially regulate cytokine production." Journal of leukocyte biology vol. 97,5 (2015): 901-908. doi: https://doi.org/10.1189/jlb.2A1014-471RR
Nikolic DM, Vadali S, He B, Ware J, Kelly T, Post SR. Prostaglandins produced during class A scavenger receptor-mediated macrophage adhesion differentially regulate cytokine production. J Leukoc Biol. 2015 May;97(5):901-908. doi: 10.1189/jlb.2A1014-471RR. Epub 2015 Feb 25. PMID: 25717147; PMCID: PMC8454924.
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Retraction.

Journal of leukocyte biology

[No authors listed]
PMID: 21357248
J Leukoc Biol. 2011 Mar;89(3):489. doi: 10.1189/jlb.0808470ret.

No abstract available.

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Retraction. J Leukoc Biol. 2011;89(3):489doi: 10.1189/jlb.0808470ret.
(2011). Retraction. Journal of leukocyte biology, 89(3), 489. https://doi.org/10.1189/jlb.0808470ret
"Retraction." Journal of leukocyte biology vol. 89,3 (2011): 489. doi: https://doi.org/10.1189/jlb.0808470ret
Retraction. J Leukoc Biol. 2011 Mar;89(3):489. doi: 10.1189/jlb.0808470ret. PMID: 21357248.
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Erratum.

Journal of leukocyte biology

[No authors listed]
PMID: 29360165
J Leukoc Biol. 2012 Feb;91(2):347. doi: 10.1189/jlb.1108702err.

No abstract available.

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Erratum. J Leukoc Biol. 2012;91(2):347doi: 10.1189/jlb.1108702err.
(2012). Erratum. Journal of leukocyte biology, 91(2), 347. https://doi.org/10.1189/jlb.1108702err
"Erratum." Journal of leukocyte biology vol. 91,2 (2012): 347. doi: https://doi.org/10.1189/jlb.1108702err
Erratum. J Leukoc Biol. 2012 Feb;91(2):347. doi: 10.1189/jlb.1108702err. PMID: 29360165.
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Erratum.

Journal of leukocyte biology

[No authors listed]
PMID: 29360166
J Leukoc Biol. 2012 Jan;91(1):182. doi: 10.1189/jlb.0211105.err.

No abstract available.

Cite
Erratum. J Leukoc Biol. 2012;91(1):182doi: 10.1189/jlb.0211105.err.
(2012). Erratum. Journal of leukocyte biology, 91(1), 182. https://doi.org/10.1189/jlb.0211105.err
"Erratum." Journal of leukocyte biology vol. 91,1 (2012): 182. doi: https://doi.org/10.1189/jlb.0211105.err
Erratum. J Leukoc Biol. 2012 Jan;91(1):182. doi: 10.1189/jlb.0211105.err. PMID: 29360166.
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Addendum.

Journal of leukocyte biology

[No authors listed]
PMID: 29360175
J Leukoc Biol. 2012 Jun;91(6):1003. doi: 10.1189/jlb.0211067.add.

No abstract available.

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Addendum. J Leukoc Biol. 2012;91(6):1003doi: 10.1189/jlb.0211067.add.
(2012). Addendum. Journal of leukocyte biology, 91(6), 1003. https://doi.org/10.1189/jlb.0211067.add
"Addendum." Journal of leukocyte biology vol. 91,6 (2012): 1003. doi: https://doi.org/10.1189/jlb.0211067.add
Addendum. J Leukoc Biol. 2012 Jun;91(6):1003. doi: 10.1189/jlb.0211067.add. PMID: 29360175; PMCID: PMC6607992.
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Platelet binding to monocytes increases the adhesive properties of monocytes by up-regulating the expression and functionality of β.

Journal of leukocyte biology

Martins PADC, van Gils JM, Mol A, Hordijk PL, Zwaginga JJ.
PMID: 29350799
J Leukoc Biol. 2006 Mar;79(3):499-507. doi: 10.1189/jlb.0605318.

Human monocytes adhere to activated platelets, resulting in the formation of platelet-monocyte complexes (PMC). Complex formation depends on the interaction between platelet-displayed P-selectin and the specific ligand for P-selectin on leukocytes, P-selectin glycoprotein ligand-1 (PSGL-1). We have recently shown...

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Martins PADC, van Gils JM, Mol A, et al. Platelet binding to monocytes increases the adhesive properties of monocytes by up-regulating the expression and functionality of β. J Leukoc Biol. 2006;79(3):499-507doi: 10.1189/jlb.0605318.
Martins, P. A. D. C., van Gils, J. M., Mol, A., Hordijk, P. L., & Zwaginga, J. J. (2006). Platelet binding to monocytes increases the adhesive properties of monocytes by up-regulating the expression and functionality of β. Journal of leukocyte biology, 79(3), 499-507. https://doi.org/10.1189/jlb.0605318
Martins, Paula A da Costa, et al. "Platelet binding to monocytes increases the adhesive properties of monocytes by up-regulating the expression and functionality of β." Journal of leukocyte biology vol. 79,3 (2006): 499-507. doi: https://doi.org/10.1189/jlb.0605318
Martins PADC, van Gils JM, Mol A, Hordijk PL, Zwaginga JJ. Platelet binding to monocytes increases the adhesive properties of monocytes by up-regulating the expression and functionality of β. J Leukoc Biol. 2006 Mar;79(3):499-507. doi: 10.1189/jlb.0605318. PMID: 29350799.
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Interview with Dr. Stephen J. McSorley and Ms. Aparna Srinivasan regarding Pivotal Advance: Secondary exposure to LPS suppresses CD4+ T cells and exacerbates murine typhoid.

Journal of leukocyte biology

Rosenberg HF.
PMID: 29350801
J Leukoc Biol. 2007 Feb;81(2):401-402. doi: 10.1189/jlb.1306194.

No abstract available.

Cite
Rosenberg HF. Interview with Dr. Stephen J. McSorley and Ms. Aparna Srinivasan regarding Pivotal Advance: Secondary exposure to LPS suppresses CD4+ T cells and exacerbates murine typhoid. J Leukoc Biol. 2007;81(2):401-402doi: 10.1189/jlb.1306194.
Rosenberg, H. F. (2007). Interview with Dr. Stephen J. McSorley and Ms. Aparna Srinivasan regarding Pivotal Advance: Secondary exposure to LPS suppresses CD4+ T cells and exacerbates murine typhoid. Journal of leukocyte biology, 81(2), 401-402. https://doi.org/10.1189/jlb.1306194
Rosenberg, Helene F. "Interview with Dr. Stephen J. McSorley and Ms. Aparna Srinivasan regarding Pivotal Advance: Secondary exposure to LPS suppresses CD4+ T cells and exacerbates murine typhoid." Journal of leukocyte biology vol. 81,2 (2007): 401-402. doi: https://doi.org/10.1189/jlb.1306194
Rosenberg HF. Interview with Dr. Stephen J. McSorley and Ms. Aparna Srinivasan regarding Pivotal Advance: Secondary exposure to LPS suppresses CD4+ T cells and exacerbates murine typhoid. J Leukoc Biol. 2007 Feb;81(2):401-402. doi: 10.1189/jlb.1306194. PMID: 29350801.
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Errata.

Journal of leukocyte biology

[No authors listed]
PMID: 29350820
J Leukoc Biol. 2006 Aug;80(2):448. doi: 10.1189/jlb.80.2.448.

No abstract available.

Cite
Errata. J Leukoc Biol. 2006;80(2):448doi: 10.1189/jlb.80.2.448.
(2006). Errata. Journal of leukocyte biology, 80(2), 448. https://doi.org/10.1189/jlb.80.2.448
"Errata." Journal of leukocyte biology vol. 80,2 (2006): 448. doi: https://doi.org/10.1189/jlb.80.2.448
Errata. J Leukoc Biol. 2006 Aug;80(2):448. doi: 10.1189/jlb.80.2.448. PMID: 29350820.
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Interview with Dr. Paul Guyre and Mr. Lehn Weaver regarding Pivotal Advance: Activation of cell surface Toll-like receptors causes shedding of the hemoglobin scavenger receptor CD163.

Journal of leukocyte biology

Rosenberg HF.
PMID: 29350816
J Leukoc Biol. 2006 Jul;80(1):24-25. doi: 10.1189/jlb.1306756.

No abstract available.

Cite
Rosenberg HF. Interview with Dr. Paul Guyre and Mr. Lehn Weaver regarding Pivotal Advance: Activation of cell surface Toll-like receptors causes shedding of the hemoglobin scavenger receptor CD163. J Leukoc Biol. 2006;80(1):24-25doi: 10.1189/jlb.1306756.
Rosenberg, H. F. (2006). Interview with Dr. Paul Guyre and Mr. Lehn Weaver regarding Pivotal Advance: Activation of cell surface Toll-like receptors causes shedding of the hemoglobin scavenger receptor CD163. Journal of leukocyte biology, 80(1), 24-25. https://doi.org/10.1189/jlb.1306756
Rosenberg, Helene F. "Interview with Dr. Paul Guyre and Mr. Lehn Weaver regarding Pivotal Advance: Activation of cell surface Toll-like receptors causes shedding of the hemoglobin scavenger receptor CD163." Journal of leukocyte biology vol. 80,1 (2006): 24-25. doi: https://doi.org/10.1189/jlb.1306756
Rosenberg HF. Interview with Dr. Paul Guyre and Mr. Lehn Weaver regarding Pivotal Advance: Activation of cell surface Toll-like receptors causes shedding of the hemoglobin scavenger receptor CD163. J Leukoc Biol. 2006 Jul;80(1):24-25. doi: 10.1189/jlb.1306756. PMID: 29350816.
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Showing 1 to 12 of 290 entries